Talk:Interleukin 31
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Wiki Education Foundation-supported course assignment
[edit]
This article is or was the subject of a Wiki Education Foundation-supported course assignment. Further details are available on the course page. Student editor(s): Immcarle48.
Above undated message substituted from Template:Dashboard.wikiedu.org assignment by PrimeBOT (talk) 00:37, 17 January 2022 (UTC)
WOT
[edit]wot? —Preceding unsigned comment added by 84.87.84.115 (talk) 22:35, 30 March 2008 (UTC)
Sources
[edit]I am interested in adding more information to this page. From my research, there appears to be a good amount of information about IL-31 ranging from its function to how it is being studied clinically. I'd like to expand the lead , function, and clinical significance sections. I may also add a section depending on what I find in my research. I'm also considering adding an image to the page.
These are my sources so far:
- Zhang Q, Putheti P, Zhou Q, Liu Q, Gao W (2008). "Structures and biological functions of IL-31 and IL-31 receptors". review. Cytokine & Growth Factor Reviews. 19 (5–6): 347–56. doi:10.1016/j.cytogfr.2008.08.003. PMC 2659402. PMID 18926762.
- Rabenhorst A, Hartmann K (2014). "Interleukin-31: a novel diagnostic marker of allergic diseases". review. Current Allergy and Asthma Reports. 14 (4): 423. doi:10.1007/s11882-014-0423-y. PMID 24510535.
- Saleem MD, Oussedik E, D'Amber V, Feldman SR (2017). "Interleukin-31 Pathway and its Role in Atopic Dermatitis: A Systematic Review". review. The Journal of Dermatological Treatment: 1–28. doi:10.1080/09546634.2017.1290205. PMID 28145790.
- Dillon SR, Sprecher C, Hammond A, Bilsborough J, Rosenfeld-Franklin M, Presnell SR, Haugen HS, Maurer M, Harder B, Johnston J, Bort S, Mudri S, Kuijper JL, Bukowski T, Shea P, Dong DL, Dasovich M, Grant FJ, Lockwood L, Levin SD, LeCiel C, Waggie K, Day H, Topouzis S, Kramer J, Kuestner R, Chen Z, Foster D, Parrish-Novak J, Gross JA (2004). "Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice". review. Nature Immunology. 5 (7): 752–60. doi:10.1038/ni1084. PMID 15184896.
- Cornelissen C, Lüscher-Firzlaff J, Baron JM, Lüscher B (2012). "Signaling by IL-31 and functional consequences". review. European Journal of Cell Biology. 91 (6–7): 552–66. doi:10.1016/j.ejcb.2011.07.006. PMID 21982586.
If anyone has anything to add or suggestions, I'd love to hear from you.Immcarle48 (talk) 05:19, 10 February 2017 (UTC)
- Looks good. All the sources except #4 are review articles. PMID 24510535 is a review and is more up-to-date than #4. Boghog (talk) 21:40, 11 February 2017 (UTC)
dorsal root ganglia
[edit]The source used for the information regarding increased expression in dorsal root ganglia (DRG) 'of different tissues' seems to have a misunderstanding of its own source for that information that made its way into this article. (https://www.jacionline.org/article/S0091-6749(05)02327-4/fulltext) (discussion paragraph 7) The DRG are independent structures of spinal nerves adjacent to the spinal cord. They contain nerve fibers that send pain, itch and other signals to the brain. The text here seems to imply that they are distributed throughout many tissues in the body which is incorrect. Should the source here be replaced with sonkoly et al? Lootnplunder (talk) 03:29, 4 May 2025 (UTC)