Talk:Autism and memory

Wiki Education Foundation-supported course assignment

This article was the subject of a Wiki Education Foundation-supported course assignment, between 20 April 2020 and 20 July 2020. Further details are available on the course page. Student editor(s): Kaylamortensen.

Above undated message substituted from Template:Dashboard.wikiedu.org assignment by PrimeBOT (talk) 17:42, 17 January 2022 (UTC)

Why this article was moved into mainspace

This article was developed as a student editing project. It was then submitted to the "Articles for Creation" process, where an editor reviewed it and rejected it. As a long-time editor and maintainer of WikiProject Neuroscience, I am convinced that the reason for rejecting it was incorrect, so I have taken the responsibility of moving the article into mainspace. It still needs development, but the topic is valid, and the material really does not belong in our main articles on autism or memory. Looie496 (talk) 06:03, 17 April 2013 (UTC)

Wiki Education assignment: Information Literacy and Scholarly Discourse

This article was the subject of a Wiki Education Foundation-supported course assignment, between 4 March 2024 and 30 April 2024. Further details are available on the course page. Student editor(s): Sneugene (article contribs). Peer reviewers: Ncrojas13.

— Assignment last updated by Kmdavis7 (talk) 18:04, 8 April 2024 (UTC)

Is the statement "The views of autism as an amnesia of memory have now been rejected." an unexplained paradigm?

Autism#Classification#ICD cites the ICD definition whose first sentence describes autism as characterised

1. by persistent deficits in the ability to initiate and to sustain reciprocal social interaction and social communication, and
2. by a range of restricted, repetitive, and inflexible patterns of behaviour, interests or activities that are clearly atypical or excessive for the individual's age and sociocultural context.

One possible explanation of these deficits is an inability of the memory to recall sufficient experiences of such socialisation, i.e. an amnesia. Also one possible explanation for these restricted patterns is an inability of the memory to recall suitably unrestricted behaviour patterns, i.e. an amnesia. The article hyperfocuses on distinguishing types of memories by differences of tasks that require memorizing but considering that memory deficits could cause autism also requires distinguishing when which type of memory stores (or transmits in between) informations consequently in time which implies such a sequence of possible deficits of memory types:

• Amnesia type 1: Deficit of a not conscious first perceptive identification, examples are cortical blindness, agnosopsia
• Amnesia type 2: Deficit of the transmission of perceptive identification into the conscious short-term memory
• Amnesia type 3: Bradyphrenia-alike deficit of the conscious short-term memory
• Amnesia type 4: Deficit of the construction of specific request patterns to the long-term memory, as in prosopagnosia at which constructing the request pattern face is insufficient and hence cannot be compared with past memories
• Amnesia type 5: Deficit of saving in the long-term memory as in anterograde amnesia
• Amnesia type 6: Deficit of the long-term memory as in retrograde amnesia
• Amnesia type 7: Deficit of the access of the long-term memory to the short-term memory for replyingly providing past memories which could happen at most subconscious. If this access does not happen in deficit but inadequate so that it distracts due to amount of long-term infos (and misleads to compensating hyperactivity) as in ADHD it would be rather a dysmnesia type 7 or even hypermnesia type 7.

So if a not totally non- but rather subconscious first perceptive identification happens but then its transmission fails as in amnesia type 2 then the short-term memory could experience a frustratingly awaiting condition to which a tendency to stimulate this transmission by such repetitive patterns could result. Also the article special interests defines them as an autism symptom group so profoundly separated from this memory symptom group that they could also likewisely appear in short-term memory as a result of hypermnesia type 7 and distract that way the memory into an autistic functioning, so that ascertaining which of it causes which appears to be a necessary part of diagnosing autism, so that just "rejecting" the "view" of autism as an amnesia appears very inconclusive here and by far does not seem to meet current efforts of profound diagnosing also by using modern research tools like ABIDE ( http://fcon_1000.projects.nitrc.org/indi/abide/ ). E.g. amygdala and hippocampus functionality investigation for such ascertaining is begun in studies like

• http://pubmed.ncbi.nlm.nih.gov/35710036/ (Altered resting state dynamic functional connectivity of amygdala subregions in patients with autism spectrum disorder: A multi-site fMRI study),
• http://pubmed.ncbi.nlm.nih.gov/38070748/ (Abnormal functional connectivity of the reward network is associated with social communication impairments in autism spectrum disorder: A large-scale multi-site resting-state fMRI study) and
• http://pubmed.ncbi.nlm.nih.gov/38576034/ (Meta-Analysis Systematic review and meta-analysis: multimodal functional and anatomical neural alterations in autism spectrum disorder),

but studies like

• http://pubmed.ncbi.nlm.nih.gov/31334941/ (Kluver-Bucy Syndrome) or
• http://pubmed.ncbi.nlm.nih.gov/34377704/ (Kluver-Bucy Syndrome: A Rare Complication of Herpes Simplex Encephalitis)

address more severe amygdala impairments also with autism-alike symptoms. Are brain imagings from there also included in ABIDE? Was ever thought about making a study that compares such findings-of-localized-brain-impairments cases with autism cases to ascertain whether such cases can tend to appear rather like autism and vice versa? --LimbicLens (talk) 23:10, 8 October 2025 (UTC)

Wiki Education assignment: Senior Capstone Seminar in Psychology

This article was the subject of a Wiki Education Foundation-supported course assignment, between 3 June 2026 and 23 July 2026. Further details are available on the course page. Student editor(s): Madisonmoss, Ccollette254 (article contribs). Peer reviewers: Ninacwills.

— Assignment last updated by Ashaa26 (talk) 23:47, 2 July 2026 (UTC)