// Workers AI · dad joke modeWhat did chymotrypsinogen B2 say? "I'm a cutting enzyme.

CTRB2
Identifiers
Aliases	CTRB2, chymotrypsinogen B2
External IDs	MGI: 88559; GeneCards: CTRB2
Enzyme activity
Gene location (Human)
Chr.	Chromosome 16 (human)
End	75,207,161 bp
Gene location (Mouse)
Chr.	Chromosome 8 (mouse)
End	112,417,642 bp
RNA expression pattern
	Top expressed in
	body of pancreas; ; islet of Langerhans; ; gonad; ; fundus; ; right coronary artery; ; duodenum; ; ectocervix; ; left uterine tube; ; placenta; ; canal of the cervix;
	Top expressed in
	pyloric antrum; ; islet of Langerhans; ; migratory enteric neural crest cell; ; cardiac muscle tissue of left ventricle; ; plantaris muscle; ; extensor digitorum longus muscle; ; interventricular septum; ; duodenum; ; corneal stroma; ; sexually immature organism;
	More reference expression data
	n/a
Gene ontology
Molecular function	peptidase activity; hydrolase activity; serine-type endopeptidase activity; serine-type peptidase activity;
Cellular component	extracellular region; extracellular space;
Biological process	digestion; cobalamin metabolic process; extracellular matrix disassembly; proteolysis;
	Sources:Amigo / QuickGO
Orthologs
Databases	NCBI: entry; OMA: entry
	440387
	66473
	ENSG00000168928; ENSG00000284810
	ENSMUSG00000031957
	Q6GPI1
	Q9CR35
	NM_001025200
	NM_025583
	NP_001020371
	NP_079859
	Wikidata
View/Edit Human	View/Edit Mouse

Chymotrypsinogen B2 is a protein that in humans is encoded by the CTRB2 gene.^[5]

References

1 2 3 ENSG00000284810 GRCh38: Ensembl release 89: ENSG00000168928, ENSG00000284810 – Ensembl, May 2017
1 2 3 GRCm38: Ensembl release 89: ENSMUSG00000031957 – Ensembl, May 2017
↑ "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
↑ "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
↑ "Entrez Gene: Chymotrypsinogen B2". Retrieved 2018-04-17.

This article on a gene on human chromosome 16 is a stub. You can help Wikipedia by adding missing information.