H2BFM

H2BW2
Identifiers
Aliases	H2BW2, H2B histone family member M, H2B.W histone 2, H2BM, H2BFM
External IDs	MGI: 1916639; GeneCards: H2BW2
Gene location (Human)
Chr.	X chromosome (human)
End	104,042,454 bp
Gene location (Mouse)
Chr.	X chromosome (mouse)
End	135,829,125 bp
RNA expression pattern
	Top expressed in
	testicle; ; cerebellar hemisphere; ; right hemisphere of cerebellum; ; ganglionic eminence; ; right ovary; ; ventricular zone; ; left ovary; ; stromal cell of endometrium; ; canal of the cervix; ; Descending thoracic aorta;
	Top expressed in
	spermatid; ; spermatocyte;
	More reference expression data
	n/a
Gene ontology
Molecular function	protein heterodimerization activity; DNA binding;
Cellular component	nucleosome; nucleus; chromosome;
Biological process	nucleosome assembly;
	Sources:Amigo / QuickGO
Orthologs
Databases	NCBI: entry; OMA: entry
	286436
	69389
	ENSG00000101812
	ENSMUSG00000048155
	P0C1H6
	Q9DAB5
	NM_001164416; NM_001377073; NM_001388464
	NM_027067
	NP_001157888; NP_001364002
	NP_081343
	Wikidata
View/Edit Human	View/Edit Mouse

H2B histone family, member M is a protein that in humans is encoded by the H2BFM gene.^[5]

References

1 2 3 GRCh38: Ensembl release 89: ENSG00000101812 – Ensembl, May 2017
1 2 3 GRCm38: Ensembl release 89: ENSMUSG00000048155 – Ensembl, May 2017
↑ "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
↑ "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
↑ "Entrez Gene: H2B histone family, member M". Retrieved 2012-03-12.

Niture SK, Doneanu CE, Velu CS, Bailey NI, Srivenugopal KS (December 2005). "Proteomic analysis of human O6-methylguanine-DNA methyltransferase by affinity chromatography and tandem mass spectrometry". Biochemical and Biophysical Research Communications. 337 (4): 1176–84. doi:10.1016/j.bbrc.2005.09.177. PMID 16226712.

This article on a gene on the human X chromosome and/or its associated protein is a stub. You can help Wikipedia by adding missing information.